Under normal physiology, tears produced by the lacrimal gland spread across the ocular surface with each blink, then drain through two tiny punctal openings at the medial corner of each eyelid. From the puncta, tears travel through the canaliculi, accumulate in the lacrimal sac, and then flow down the nasolacrimal duct to drain into the nasal cavity beneath the inferior turbinate. When any part of this pathway becomes obstructed, the tear drainage is impaired and tears overflow onto the face.
In newborns, the most common cause is failure of the thin membrane at the lower end of the nasolacrimal duct (Hasner's valve) to open fully before or shortly after birth. This membranous obstruction accounts for the majority of congenital watery eye presentations and frequently resolves with conservative management in the first year of life. In adults, blockage may result from infection-related scarring, inflammation, age-related duct narrowing, trauma, nasal or sinus disease, or tumour — each requiring different diagnostic and therapeutic approaches as evaluated by an eye specialist in Delhi.
Untreated or long-standing blocked tear duct creates a stagnant pool of tears within the lacrimal sac — an environment susceptible to bacterial proliferation. This can progress to acute dacryocystitis, a painful infection of the lacrimal sac that may require urgent antibiotics and eventual surgical drainage.